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Researchers have uncovered new evidence regarding how the genetic factor linked to Alzheimer's disease has evolved, specifically the A.β.O.E. 4 gene, and ways to slow its harmful effect on the brain. It has been shown that this gene damages brain blood vessels and promotes the accumulation of abnormal proteins, leading to deterioration of blood flow and brain damage. Studies indicate that inhibiting TGF-beta protein, which is involved in tissue repair, can protect blood vessels and halt their deterioration associated with the gene. This discovery opens new prospects for treatments aimed at maintaining blood vessel function and reducing amyloid buildup in Alzheimer's disease.
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