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Alzheimer's Risk May Start Outside Brain
A new study suggests that Alzheimer's disease is not limited to brain damage alone but also involves immune responses originating outside the brain. By directing T cells through traditional dendritic cells, a specific type of T cell (CD8+) can reach the brain, contributing to neuronal damage and the accumulation of the "Tau" protein. Experiments showed that inhibiting the protective cDC1 cells, which assist in activating T cells, reduces markers of neurodegeneration, without changing Tau protein levels. This indicates that the immune response may be a key driver of damage in Alzheimer's disease, and that the lymph nodes in the neck play an important role in the immune activation associated with the condition.
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