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الشروق التونسية
الشروق التونسية
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A research team from Chiba University in Japan has discovered that some drug-resistant cells in tumors harboring KRAS mutations rely on glutamine metabolism and utilize lysosomes to survive. When glutamine metabolism and lysosomal function are inhibited alongside KRAS inhibitor treatment, the number of resistant cells decreases, paving the way for the development of a comprehensive therapy that could eliminate all cancer cells and reduce the likelihood of tumor recurrence.
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