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A new study has revealed that autoantibodies play a key role in the appearance of neurological symptoms in some long COVID-19 patients, as they interacted more extensively with nerve tissue in these individuals. Experiments showed that these antibodies are associated with issues such as loss of taste and smell, nausea, and joint pain. Additionally, injecting them into mice led to increased pain sensitivity, balance disturbances, and fatigue. The findings suggest that targeting autoantibodies could open new therapeutic avenues for long COVID patients suffering from neurological symptoms.
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